Summer is Over – Why They Say We Need Vitamin D Now
England is the country of bad weather, so they say. Why we tend to get sick when temperatures drop is not only because of the cold breezes. The lack of sun has an even more significant impact on our health. Vitamin D is the substance that contributes significantly to the stability of our immune system and makes important biochemical processes possible. Without vitamin D, serious diseases such as rickets can arise.
Unlike other vitamins, the human body can produce vitamin D by itself from a precursor, which is why the name “vitamin” is controversial. The precursor is formed from cholesterol, which is abundant in the body under normal conditions. The help of sunlight converts the precursor into a transportable form of the vitamin that can be processed by the human body.
Through the skin, 80-90% of vitamin D is provided. Nevertheless, a small part of this vitamin can be absorbed by means of specific foods. Especially in fish, mushrooms and dairy products there are small amounts of the vitamin, but in a different form. In food there is vitamin D2 and not the effective D3.
What role does vitamin D play in the human body?
Studies have shown a strong correlation between deficiencies of vitamin D and the presence of many chronic diseases: osteomalacia and rickets, high blood pressure as well as diabetes and allergies.
Vitamin D supports the absorption of calcium in the body. This ensures that there is always enough calcium for vital metabolic functions such as the transmission of nerve signals and muscle work. The more calcium the body can absorb with the help of vitamin D, the more it can be stored as a reserve in the skeleton. Vitamin D is used, for example, to form the enzyme osteocalcin that binds calcium and stores it in the bones. Thus, vitamin D also promotes the structure and strength of the bones. Vitamin D deficiencies lead to rickets with children. A lack of calcium in the bones can cause growth disorders, malformations and bone deformations. You can look up our vitamin D products here.
Vitamin D and other nutrients
When supplementing vitamin D3, it is important to keep an eye on the nutrients that are involved in the biochemical processes related to absorption, conversion and effects of vitamin D. These include the following nutrients:
Vitamin D3 has the important task of regulating the blood calcium level. It regulates e.g. the formation of the calcium-binding enzyme osteocalcin. However, this must be activated first, which happens through vitamin K. Without vitamin K2, there is a risk of calcification in tissue (hypercalcaemia). Therefore, a sufficient vitamin K intake prevents coronary artery diseases and heart attacks. Shop vitamin K now.
Magnesium is needed to enable the utilization of vitamin D and convert it into its active form. The higher the intake of vitamin D, the higher the magnesium consumption in the body. Many side effects caused by high doses of vitamin D, such as headaches, heart stuttering and muscle spasms, are often the result of a magnesium deficiency through vitamin D. Shop magnesium now.
The vitamin D receptor, found in almost all cells and responsible for the action of vitamin D, has two zinc molecules at its base. Therefore, a strong zinc deficiency can limit the vitamin D function because the body cannot form the corresponding receptors. Shop zinc now.
W. Hollis: Circulating 25-Hydroxyvitamin D Levels Indicative of Vitamin D Sufficiency: Implications for Establishing a New Effective Dietary Intake Recommendation for Vitamin D. In: J Nutr. Band 135(2), 2005, S. 317–322.
Kipshoven, Christoph. Querschnittsstudie zur Abschätzung des Vitamin-D-Status in der Bevölkerung in Deutschland (DEVID-Studie). Diss. Köln, Univ., Diss., 2010, 2010.
Fetahu IS, Höbaus J, Kállay E. Vitamin D and the epigenome. Frontiers in Physiology. 2014; 5:164. doi:10.3389/fphys.2014.00164.
Biesalski, Hans K. Vitamin D recommendations–beyond deficiency. Annals of Nutrition and Metabolism, 2011, 59. Jg., Nr. 1, S. 10-16.
ZIPITIS CS, AKOBENG AK., VITAMIN D SUPPLEMENTATION IN EARLY CHILDHOOD AND RISK OF TYPE 1 DIABETES: A SYSTEMATIC REVIEW AND META-ANALYSIS. Arch Dis Child, 2008; 93 (6): 517-517